Neuroanatomical distribution of receptors for a novel voltage-sensitive calcium-channel antagonist, SNX-230 (ω-conopeptide MVIIC)
Reference (30)
- et al.
Subunit structure and localization of dihydropyridine-sensitive calcium channels on mammalian brain, spinal cord and retina
Neuron
(1990) - et al.
OmegaConus geographus toxin: a peptide that blocks calcium channels
FEBS Lett.
(1987) - et al.
Autoradiographic localization of calcium channel antagonist receptors in rat brain with [3H]-nitrendipine
Brain Res.
(1985) - et al.
A newConus peptide ligand for mammalian presynaptic Ca2+ channels
Neuron
(1992) - et al.
Autoradiographic localization of calcium channels with [125I]-ω-conotoxin in rat brain
Eur. J. Pharmacol.
(1988) - et al.
Distinctive biophysical and pharmacological properties of class A (B1) calcium channelα1 subunits
Neuron
(1993) - et al.
Characteristics of [125I]-omega conotoxin MVIIA binding to rat neocortical membranes
Neurosci. Lett.
(1993) - et al.
Autoradiographic visualization in rat brain of receptors for ω-conotoxin GVIA, a newly discovered calcium antagonist
Brain Res.
(1988) - et al.
Molecular diversity of voltage dependent Ca2+ channels
Trends Pharmacol. Sci.
(1991) - et al.
Biochemical properties and subcellular distribution of an N-type calcium channelα1 subunit
Neuron
(1992)
Pharmacological properties and localization of calcium channel types in the cerebellar cortex
Soc. Neurosci. Abstr.
(1993)
Cited by (117)
Upregulation of N-type calcium channels in the soma of uninjured dorsal root ganglion neurons contributes to neuropathic pain by increasing neuronal excitability following peripheral nerve injury
2018, Brain, Behavior, and ImmunityCitation Excerpt :At present, the therapeutic effect is explained by the fact that Cav2.2 channels are expressed in presynaptic terminals, and opening the channel subtype is essential for release of neurotransmitters such as glutamate, substance P and CGRP (see (Perret and Luo, 2009) for a review). While Cav2.2 channels are also expressed in the soma of DRG neurons (Gohil et al., 1994; Kerr et al., 1988), their function is largely unknown. In the present study, we found that Cav2.2 protein was upregulated in the soma of uninjured DRG neurons in both L5-SNL and L5-VRT models of neuropathic pain (Figs. 1–3).
N- and L-type calcium channels blocker cilnidipine ameliorates neuropathic pain
2016, European Journal of PharmacologyMechanisms of conotoxin inhibition of N-type (Ca<inf>v</inf>2.2) calcium channels
2013, Biochimica et Biophysica Acta - BiomembranesAntihypertensives
2024, Pharmacology in Veterinary Anesthesia and Analgesia
Copyright © 1994 Published by Elsevier B.V.