Regular paperα1-Adrenoceptor subtypes mediating antinatriuresis in Wistar and stroke-prone spontaneously hypertensive rats
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2013, Handbook of Clinical NeurologyCitation Excerpt :Later reports which attempted to examine in greater depth the subtypes of α1-adrenoceptors involved in meditating the renal nerve-induced reductions in renal hemodynamics found that a large component required activation of the α1A-adrenoceptor subtype (Witkowski et al., 2011). At the level of the tubular epithelia cells, a number of studies provided evidence that the antinatriuresis resulting from low-level stimulation of the renal sympathetic nerves could be blocked by prazosin, but not by the α2-adrenoceptor antagonist idazoxan, consistent with the involvement of α1-adrenoceptors (Johns and Manitius, 1986) which was later shown (Sattar and Johns, 1995) to primarily involve α1A-adrenoceptors. It became evident at a very early stage that renal nerve-dependent renin release involved the activation of β-adrenoceptors.
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