Nitric oxide production in experimental alcoholic liver disease in the rat: Role in protection from injury☆
References (62)
- et al.
Endothelium-derived relaxing factor: identification as nitric oxide and role in the control of vascular tone and platelet function
Biochem Pharmacol
(1988) - et al.
Dietary linoleic acid is required for development of experimentally induced alcoholic liver injury
Life Sci
(1989) - et al.
Plasma endothelin levels in chronic ethanol-fed rats: relationship to pathologic liver injury
Life Sci
(1994) - et al.
Decreased prostacyclin production by liver non-parenchymal cells precedes liver injury in experimental alcoholic liver disease
Life Sci
(1994) - et al.
Clearance of gut derived endotoxins by the liver: release and modification of 3H, 14C lipopolysaccharide by isolated rat Kupffer cells
Gastroenterology
(1989) - et al.
Analysis of nitrate, nitrite, and [15N] nitrate in biological fluids
Anal Biochem
(1982) - et al.
Phorbol esters induce nitric oxide synthase activity in rat hepatocytes: antagonism with the induction elicited by lipopolysaccharide
J Biol Chem
(1992) - et al.
Nitric oxide kills hepatocytes by mobilizing mitochondrial calcium
Biochem Biophys Res Commun
(1994) - et al.
Peroxynitrite-induced membrane lipid peroxidation: the cytotoxic potential of superoxide and nitric oxide
Arch Biochem Biophys
(1991) The delicate balance of nitric oxide and superoxide in liver pathology
Gastroenterology
(1995)
Endotoxin stimulated nitric oxide production increases liver injury and reduces rat liver chemiluminescence during reperfusion
Gastroenterology
(1995)
Nitric oxide: a novel biological messenger
Cell
(1992)
Nitric oxide as an antioxidant
Arch Biochem Biophys
(1991)
Redox signaling-nitrosylation and related target interactions of nitric oxide
Cell
(1994)
Metabolic alterations produced in the liver by chronic ethanol administration: changes related to energy parameters of the cell
Biochem J
(1973)
Metabolic alterations produced in the liver by chronic ethanol administration: increased oxidative capacity
Biochem J
(1973)
The swift increase in alcohol metabolism: time course for increase in hepatic oxygen uptake and the involvement of glycolysis
Biochem J
(1980)
Incomplete compensation of enhanced hepatic oxygen consumption in rats with alcoholic centrilobular necrosis
Hepatology
(1989)
Compensatory mechanisms in response to an elevated hepatic oxygen consumption in chronically ethanol-fed rats
Am J Physiol
(1985)
Effects of intravenous infusions of ethanol upon estimated hepatic blood flow in man
J Clin Invest
(1954)
Biochemistry of nitric oxide and its redox-activated forms
Science
(1992)
Macrophage cytotoxicity: role for l-arginine deaminase activity and amino nitrogen oxidation to nitrite
Science
(1987)
Vascular endothelial cells synthesize nitric oxide from l-arginine
Nature
(1988)
Regulation of nitric oxide production by stimulated rat Kupffer cells
Pathobiology
(1991)
Beef fat prevents alcoholic liver disease in the rat
Alcohol Clin Exp Res
(1989)
Insights into the pathogenesis of alcoholic liver necrosis and fibrosis: status report
Hepatology
(1989)
Ethanol-induced hepatic fibrosis in the rat
Alcohol Clin Exp Res
(1986)
Protective effects of a novel quinone derivative, (2E)-3-[5-(2,3 dimethoxy-6-methyl-1,4 benzoquinoyl)]-2-nonyl-2-propenoic acid on experimental alcoholic liver injury
J Pharmacol Exp Ther
(1993)
Pathology of alcoholic liver disease
Semin Liver Dis
(1993)
Nitric oxide synthase in human and rat lung: immunocytochemical and histochemical localization
Am J Respir Cell Mol Biol
(1993)
Pitfalls in histochemical localization studies of NADPH generating enzymes or enzyme systems in rat small intestine
Histochemistry
(1972)
Cited by (0)
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Supported in part by Nitromed. Dr. Stamler is a Pew Scholar in the Biomedical Sciences and the recipient of a Clinical Investigator Award from the National Institues of Health (HL 02582).
The authors thank Dr. Peter Thomas, Robert Miller-Cassman, Vincent Bird, Vincenzo Barletta, and Marianne Slaney for assistance.
Copyright © 1995 Published by Elsevier Inc.