Original articleAtrial natriuretic peptide inhibits oxidant-induced increases in endothelial permeability
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Role of microtubules in attenuation of PepG-induced vascular endothelial dysfunction by atrial natriuretic peptide
2015, Biochimica et Biophysica Acta - Molecular Basis of DiseaseCitation Excerpt :The two major ANP receptors, NPR-A and NPR-B act as membrane-associated guanylate cyclases [9], and elevation of cGMP levels is a primary response to ANP stimulation. ANP-induced elevation of cGMP decreased basal levels of lung EC permeability, attenuated pulmonary EC barrier dysfunction caused by hydrogen peroxide [10,11], and inhibited oxidant-induced pulmonary edema observed in perfused rabbit lungs [12]. However, ANP-mediated elevation of cGMP increased lung vascular permeability in the ischemia reperfusion model of lung injury [13], suggesting context-specific effects of cGMP and ANP in different models.
Post-ischemic Infusion of Atrial Natriuretic Peptide Attenuates Warm Ischemia-Reperfusion Injury in Rat Lung
2009, Journal of Heart and Lung TransplantationNatriuretic peptides differentially attenuate thrombin-induced barrier dysfunction in pulmonary microvascular endothelial cells
2006, Experimental Cell ResearchChapter 4 Membrane and cellular signaling of integrity and acute activation
2005, Advances in Molecular and Cell BiologyChapter 15 Advances in protection of endothelial barrier function
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