Doxorubicin cardiotoxicity and the control of iron metabolism: quinone-dependent and independent mechanisms

Methods Enzymol. 2004:378:340-61. doi: 10.1016/S0076-6879(04)78025-8.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Antibiotics, Antineoplastic / toxicity*
  • Doxorubicin / toxicity*
  • Ferritins / metabolism
  • Free Radicals / metabolism
  • Heart Diseases / chemically induced*
  • Heart Diseases / physiopathology
  • Humans
  • Iron / metabolism*
  • Iron Regulatory Protein 1 / metabolism
  • Iron Regulatory Protein 2 / genetics
  • Iron Regulatory Protein 2 / metabolism
  • Models, Biological
  • Molecular Structure
  • Mutagenesis, Insertional
  • Protein Processing, Post-Translational
  • Quinones / metabolism*
  • RNA, Messenger / metabolism
  • RNA-Binding Proteins / metabolism
  • Receptors, Transferrin / metabolism
  • Response Elements / physiology
  • Structure-Activity Relationship
  • Transferrin / metabolism

Substances

  • Antibiotics, Antineoplastic
  • Free Radicals
  • Quinones
  • RNA, Messenger
  • RNA-Binding Proteins
  • Receptors, Transferrin
  • Transferrin
  • Doxorubicin
  • Ferritins
  • Iron
  • Iron Regulatory Protein 1
  • Iron Regulatory Protein 2