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Abstract

The aryl-hydrocarbon receptor, but not the aryl-hydrocarbon receptor nuclear translocator protein, is rapidly depleted in hepatic and nonhepatic culture cells exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin.

R S Pollenz
Molecular Pharmacology March 1996, 49 (3) 391-398;

Article Information

vol. 49 no. 3 391-398
PubMed 
8643077
Published By 
American Society for Pharmacology and Experimental Therapeutics
Print ISSN 
0026-895X
Online ISSN 
1521-0111
History 
  • Published online March 1, 1996.

Author Information

  1. R S Pollenz
  1. Department of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, 29425, USA.

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    Abstract

    The aryl-hydrocarbon receptor, but not the aryl-hydrocarbon receptor nuclear translocator protein, is rapidly depleted in hepatic and nonhepatic culture cells exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin.

    R S Pollenz
    Molecular Pharmacology March 1, 1996, 49 (3) 391-398;
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