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Research ArticleArticle

An N-Terminal Histidine Is the Primary Determinant of α Subunit-Dependent Cu2+ Sensitivity of αβ3γ2L GABAA Receptors

Heejeong Kim and Robert L. Macdonald
Molecular Pharmacology November 2003, 64 (5) 1145-1152; DOI: https://doi.org/10.1124/mol.64.5.1145

Article Information

vol. 64 no. 5 1145-1152
DOI 
https://doi.org/10.1124/mol.64.5.1145
PubMed 
14573764
Published By 
American Society for Pharmacology and Experimental Therapeutics
Print ISSN 
0026-895X
Online ISSN 
1521-0111
History 
  • Received April 21, 2003
  • Accepted August 4, 2003
  • Published online October 21, 2003.
Copyright & Usage 
The American Society for Pharmacology and Experimental Therapeutics

Author Information

  1. Heejeong Kim and
  2. Robert L. Macdonald
  1. Department of Neurology, University of Michigan Health System, University of Michigan, Ann Arbor, Michigan (H.K.); and Departments of Neurology, Molecular Physiology and Biophysics, and Pharmacology, Vanderbilt University, Nashville, Tennessee (R.L.M.)
  1. Address correspondence to:
    Dr. Robert L. Macdonald, Department of Neurology, Vanderbilt University, 2100 Pierce Ave., Nashville, TN 37212 (E-mail: robert.macdonald{at}vanderbilt.edu).
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An N-Terminal Histidine Is the Primary Determinant of α Subunit-Dependent Cu2+ Sensitivity of αβ3γ2L GABAA Receptors

Heejeong Kim and Robert L. Macdonald
Molecular Pharmacology November 1, 2003, 64 (5) 1145-1152; DOI: https://doi.org/10.1124/mol.64.5.1145
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