Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

doi:10.1124/mol.80.3.550

CorrectionErratum

Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

Molecular Pharmacology September 2011, 80 (3) 550; DOI: https://doi.org/10.1124/mol.80.3.550

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Molecular Pharmacology

Vol. 80, Issue 3

1 Sep 2011

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CorrectionErratum

Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

Molecular Pharmacology September 1, 2011, 80 (3) 550; DOI: https://doi.org/10.1124/mol.80.3.550

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CorrectionErratum

Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

Molecular Pharmacology September 1, 2011, 80 (3) 550; DOI: https://doi.org/10.1124/mol.80.3.550

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Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

MolPharm articles become freely available 12 months after publication, and remain freely available for 5 years.

Non-open access articles that fall outside this five year window are available only to institutional subscribers and current ASPET members, or through the article purchase feature at the bottom of the page.

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Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

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Correction to “Evidence that γ-Secretase-Mediated Notch Signaling Induces Neuronal Cell Death via the Nuclear Factor-κB-Bcl-2-Interacting Mediator of Cell Death Pathway in Ischemic Stroke”

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