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Re: Abkhezr M and Dryer SE (2014) Angiotensin II and Canonical Transient Receptor Potential-6 Activation Stimulate Release of a Signal Transducer and Activator of Transcription 3–Activating Factor from Mouse Podocytes Mol Pharmacol August 2014 86:150–158; doi:10.1124/mol.114.092536
Molecular Pharmacology September 2016, 90 (3) 160; DOI: https://doi.org/10.1124/mol.114.092536retraction
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RetractionNotice of Retraction
Re: Abkhezr M and Dryer SE (2014) Angiotensin II and Canonical Transient Receptor Potential-6 Activation Stimulate Release of a Signal Transducer and Activator of Transcription 3–Activating Factor from Mouse Podocytes Mol Pharmacol August 2014 86:150–158; doi:10.1124/mol.114.092536
Molecular Pharmacology September 1, 2016, 90 (3) 160; DOI: https://doi.org/10.1124/mol.114.092536retraction
RetractionNotice of Retraction
Re: Abkhezr M and Dryer SE (2014) Angiotensin II and Canonical Transient Receptor Potential-6 Activation Stimulate Release of a Signal Transducer and Activator of Transcription 3–Activating Factor from Mouse Podocytes Mol Pharmacol August 2014 86:150–158; doi:10.1124/mol.114.092536
Molecular Pharmacology September 1, 2016, 90 (3) 160; DOI: https://doi.org/10.1124/mol.114.092536retraction
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- Notice of Retraction: Acharya P, Engel JC, Correia MA (2009) Hepatic CYP3A suppression by high concentrations of proteasomal inhibitors: A consequence of endoplasmic reticulum (ER) stress induction, activation of RNA-dependent protein kinase-like ER-bound eukaryotic initiation factor 2α (eIF2α)-kinase (PERK) and general control nonderepressible-2 eIF2α kinase (GCN2), and global translational shutoff. Mol Pharmacol 76(3):503–515; DOI: https://doi.org/10.1124/mol.109.056002.
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