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Correction to: “Inhibition of GLI1 Expression by Targeting the CRD-BP–GLI1 mRNA Interaction Using a Specific Oligonucleotide”
Molecular Pharmacology June 2016, 89 (6) 707; DOI: https://doi.org/10.1124/mol.115.102434err
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Correction to: “Biased Type 1 Cannabinoid Receptor Signaling Influences Neuronal Viability in a Cell Culture Model of Huntington Disease”
Molecular Pharmacology June 2016, 89 (6) 694; DOI: https://doi.org/10.1124/mol.115.101980err
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Correction to “Pharmacological Discrimination of Calcitonin Receptor: Receptor Activity-Modifying Protein Complexes”
Molecular Pharmacology April 2016, 89 (4) 484; DOI: https://doi.org/10.1124/mol.115.008615err
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Correction to “α-Conotoxins Identify the α3β4* Subtype as the Predominant Nicotinic Acetylcholine Receptor Expressed in Human Adrenal Chromaffin Cells”
Molecular Pharmacology February 2016, 89 (2) 322; DOI: https://doi.org/10.1124/mol.115.100982err
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Correction to“The LRRC26 Protein Selectively Alters the Efficacy of BK Channel Activators”
Molecular Pharmacology January 2016, 89 (1) 214; DOI: https://doi.org/10.1124/mol.115.075234err
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Correction to “Desformylflustrabromine (dFBr) and [³H]dFBr-Labeled Binding Sites in a Nicotinic Acetylcholine Receptor”
Molecular Pharmacology January 2016, 89 (1) 141; DOI: https://doi.org/10.1124/mol.115.098913err
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Correction to “Synthetic metallochaperone ZMC1 rescues mutant p53 conformation by transporting zinc into cells as an ionophore”
Molecular Pharmacology December 2015, 88 (6) 1084; DOI: https://doi.org/10.1124/mol.115.097550err
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Correction to “Gα₁₃/PDZ-RhoGEF/RhoA Signaling Is Essential for Gastrin-Releasing Peptide Receptor–Mediated Colon Cancer Cell Migration”
Molecular Pharmacology November 2015, 88 (5) 880; DOI: https://doi.org/10.1124/mol.115.093914err
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Correction to: “Preclinical Evaluation of DMA, a Bisbenzimidazole, as Radioprotector: Toxicity, Pharmacokinetics, and Biodistribution Studies in Balb/c Mice”
Molecular Pharmacology October 2015, 88 (4) 813; DOI: https://doi.org/10.1124/mol.115.098376err
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Correction to “The Tumor Proteasome Is a Primary Target for the Natural Anticancer Compound Withaferin A Isolated from ‘Indian Winter Cherry’”
Molecular Pharmacology May 2015, 87 (5) 865; DOI: https://doi.org/10.1124/mol.114.030015err

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