Research paperIdentification of functional aryl hydrocarbon receptor and aryl hydrocarbon receptor nuclear translocator in murine splenocytes☆
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Regulation of Bach2 by the aryl hydrocarbon receptor as a mechanism for suppression of B-cell differentiation by 2,3,7,8-tetrachlorodibenzo-p-dioxin
2011, Toxicology and Applied PharmacologyCitation Excerpt :For the supershifts, the nuclear proteins were incubated with anti-Bach2 (L-17) antibody (sc-14704X) or normal goat IgG (sc-2028) (Santa Cruz Biotechnology, Inc., Santa Cruz, CA) for 30 min at RT prior to the addition of radiolabeled probes. Nuclear proteins for the DRE3 and Bach2 intron1 EMSA were prepared from resting CH12.LX cells treated with 10 nM TCDD or 0.02% DMSO as vehicle for 1 h as previously described (Probst et al., 1993; Williams et al., 1996) with modifications. The cells were harvested by centrifugation at 300 × g for 5 min followed by one wash with 1× HBSS and incubated in HB buffer (10 mM HEPES (pH:7.5), 1 mM MgCl2) for 10 min.
Antagonism of aryl hydrocarbon receptor-dependent induction of CYP1A1 and inhibition of IgM expression by di-ortho-substituted polychlorinated biphenyls
2003, Toxicology and Applied PharmacologyPolychlorinated biphenyl-induced apoptosis of murine spleen cells is aryl hydrocarbon receptor independent but caspases dependent
2002, Toxicology and Applied Pharmacology2,3,7,8-Tetrachlorodibenzo-p-dioxin does not directly alter the phenotype of maturing B cells in a murine coculture system
2002, Toxicology and Applied Pharmacology
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This work was supported, in part, by funds from NIEHS Grant ES03520 and NIGMS Training Grant GM07392.