Plasma and cerebrospinal fluid free amino acid concentration in post-traumatic cerebral oedema in patients with shock
References (15)
- et al.
A comparative study of free amino acids in coma of different aetiology
Resuscitation
(1975) - et al.
Examination of the metabolism of oedematous brain tissue. IV — The influence of carbon dioxide, bicarbonate and pH on respiration and aerobic glycolysis of normal and oedematous rabbit brain in vitro
Acta Neurochir.
(1974) - et al.
Signs of cerebral hypoxia in hyperventilation
Experientia
(1968) - et al.
The effect of asphyxia upon lactate, pyruvate and bicarbonate concentrations of brain tissue and cisternal CSF, upon the tissue concentrations of phosphocreatine and adenine nulceotides in anesthetized rats
Acts, Physiol. Scand.
(1970) - et al.
Plasma aminoacids in normal humans and patients with shock
Surg., Gynecol. Obstet.
(1967) Protein metabolism distribution under conditions of shock and surgical interference
Am. J. Surg.
(1936)- et al.
Studies of the plasma aminoacids and amino conjugates in patients with severe battle wounds
Surg., Gynecol. Obstet.
(1955)
There are more references available in the full text version of this article.
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