Survivin and apoptosis control
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The noncanonical function of borealin, a component of chromosome passenger complex, promotes glycolysis via stabilization of survivin in squamous cell carcinoma cells
2024, Biochemical and Biophysical Research CommunicationsSurvivin promotes a glycolytic switch in CD4<sup>+</sup> T cells by suppressing the transcription of PFKFB3 in rheumatoid arthritis
2022, iScienceCitation Excerpt :Survivin, an oncoprotein encoded by BIRC5, is widely expressed in malignancies and during renewal of nonmalignant hematopoetic cells.18,19 Cytosolic and mitochondrial localization of survivin is tightly linked to its anti-apoptotic function,20 while nuclear localization of survivin has been attributed to its role in the chromosomal passenger complex21 and in the formation of macromolecular complexes, potentially supporting gene expression.18,22,23,24 Shuttling of survivin between cytosol and nucleus is assisted by exportin 1.25,26
Effects of doxorubicin and docetaxel on susceptibility to apoptosis in high expression level of survivin in HEK and HEK-S cell lines as in vitro models
2020, Biochemical and Biophysical Research CommunicationsThe role of perilipin 2 in Pseudomonas aeruginosa pulmonary infection
2020, Respiratory Physiology and NeurobiologyCitation Excerpt :It was consistent with the results that PLIN2 silencing downregulated TOP and TOP2A expression, and reduced cell proliferation. BCL2L1 (Bcl‐xl) (Warren et al., 2019) and BIRC5 (SURVIVIN) (Altieri, 2003) are significant anti-apoptosis proteins. CDK1 is a major component of the cell cycle progression engine, contributing to the mitotic prophase and metaphase (Castedo et al., 2002).
CSN5/Jab1 facilitates non-small cell lung cancer cell growth through stabilizing survivin
2018, Biochemical and Biophysical Research CommunicationsRecent development of transition metal complexes with in vivo antitumor activity
2017, Journal of Inorganic BiochemistryCitation Excerpt :An accumulation of 5 was observed in mitochondria and the cytoplasm, but not in the nucleus or lysosome. Treatment of 5 also dose-dependently inhibit expression of survivin, an inhibitor of apoptosis [21,22] that is much higher expressed in a majority of human cancer cells [23]. 5 also acted as an activator of caspase-3 and poly (ADP-ribose) polymerase, leading to the induction of apoptosis.