Mechanisms of allergy and clinical immunologyProstaglandin D2 activates group 2 innate lymphoid cells through chemoattractant receptor-homologous molecule expressed on TH2 cells
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Supported by the Wellcome Trust (to P.K.), the Medical Research Council (to G.O.), NIHR Biomedical Research Centre Programme (to L.X., G.O., and M.S.), Oxford Martin School (to P.K.), the British Medical Association (James Trust; to G.O., P.K., and L.X.), Oxfordshire Health Services Research Committee Research Grant (to L.X.), and the National Institutes of Health (to P.K. and L.X.). P.K. is an NIHR Senior Investigator.
Disclosure of potential conflict of interest: J. M. Mjösberg has received research support from the Swedish Research Council. A. N. J. McKenzie has received research support from, has patents (planned, pending, or issued) from, and has royalties from Janssen. H. Spits has received research support from the ERC as an advanced grant; is employed by the Academic Medical Center of the University of Amsterdam; and is employed by, has patents planned, pending, or issued from, has stock/stock options in, and is a founder of AIMM therapeutics. P. Klenerman has received research support from The Wellcome Trust, the NIH, and the NIHR Biomedical Research Centre. G. Ogg has received research support from the Medical Research Council, the Biomedical Research Centre, the British Medical Association, and Janssen Pharmaceuticals and has received consultancy fees from Novartis and Lilly. The rest of the authors declare that they have no relevant conflicts of interest.
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These authors contributed equally to this work.
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These authors contributed equally to this work as joint senior authors.